The repair mechanisms of the skin are impaired as we age, and our bodies need more time to repair its protective barrier from an injury. This is due to the disruption of communication between the cells in the skin and the immune cells that repair the skin.
The epidermis relies on the dermis for its nourishment. Fibroblasts are vital to skin maintenance and healing. Fibroblasts produce collagen and elastin.
The reduction of collagen and elastin in the skin with ageing, causes the skin to become thinner. This reduction of elasticity which not only can increase the formation of wrinkles but can also make the skin easily damaged therefore vulnerable., The key immune cells in the epidermis are: Langerhans cells and Keratinocytes (skin cells). These cells become weakened with age as well as UV damage, causing enhanced oxidative stress to the skin.
Due to synthesis errors. The faulty melanocytes become larger, and pigmentation may occur in areas, mostly which are exposed to the sun. Due to the reduction in the supportive collagen. and elastin fibers, the density of the skin is greatly reduced; eventually leading to sagging of the skin as well as the contours of the face including hollows and Furrows.
Written by
Angela Blemmings
Company Director
EyeCandy Training Ltd
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